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hamsters. Some—but not all—of the hamsters The third method virologists use to prove infection and pathogenic-
got pneumonia, and some even died. Perplex- ity—the method they usually rely on—is to infect human and animal
ingly, however, some of the hamsters didn’t tissue with a “culture” of the virus to see what happens. As we have
even get sick at all, which certainly doesn’t already pointed out, this inoculation of solutions reportedly containing
square with the deadly contagious virus theory. the virus onto a variety of tissue cultures has never been shown to kill
Because there was no comparison group, we (lyse) the tissue, unless the tissue is first starved and poisoned.
also have no idea what would have happened if Nevertheless, researchers used this third approach in a study entitled,
the researchers had squirted plain lung cancer “Severe Acute Respiratory Syndrome Coronavirus 2 from Patient with
cells into the lungs of the hamsters; probably Coronavirus Disease, United States” published in the CDC’s Emerging
not anything good. Infectious Diseases journal in June 2020. The purpose of the study was
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In the mouse study, researchers infected for a group of about thirty-five virologists to describe the state of the sci-
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both transgenic mice (that is, mice genetically ence dealing with the isolation, purification and biological characteristics
programmed to get sick) and wild (normal) mice of the new SARS-CoV-2 virus, and to share this information with other
with unpurified virus. None of the wild mice ex- scientists for their own research. A thorough and careful reading of this
posed to the “virus” got sick. Of the transgenic important paper reveals some shocking findings.
mice, a statistically insignificant number either First, in the Methods section titled “Whole Genome Sequencing,” we
lost some fur luster or experienced weight loss. find that rather than having isolated the virus and sequencing the genome
Thus, scientists have not been able to show that from end to end, they “designed 37 pairs of nested PCRs spanning the
the Covid-19 “virus” causes harm to animals. genome on the basis of the coronavirus reference sequence.” What this
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MYXOMATOSIS

In response to our claim that so-called “viruses” are actually helpful exosomes—which do not cause disease but
which the body makes in response to toxins, starvation and injury—we received a chiding email.
“As a young girl, living in rural Manitoba, Canada, my brother would harvest wild rabbits for our winter protein.
When my brother was skinning the rabbits, he was extremely careful to check for bubbles under the skin and in the
meat. If he found bubbles, he knew that the rabbits had a virus, due to overpopulation. We were not able to eat
those rabbits that year. . . and would have to wait until nature had taken its course. My point being, there is no 5G
or anything that would support your theory.” The writer also describes tuberculosis (TB) among the northern Inuit,
“which had taken hold, due to living in crowded conditions and of course eating the foods that the Europeans had
introduced to them. Killing people off with fast foods and the like has brought us to where we are. This Covid virus
is just the beginning of the major viruses that are just around the corner.” The email’s author also referred to very
crowded and filthy living conditions in China, presumably in connection with diseases like cholera.
The rabbit disease to which she refers is called myxomatosis. The official view is that a virus carried in the sa-
liva of fleas, mosquitoes and other insects causes the disease, with overcrowded rabbit populations being the most
vulnerable to illness. Symptoms include swelling at the site of the “infection”—the insect bite—followed by fever,
swelling in other areas (eyelids, face, base of ears, anogenital area), skin lesions, ocular and nasal discharge, respira-
tory distress, hypothermia, closure of the eyelids due to swelling, and death. These are some very miserable rabbits!
A typical study cited for a viral cause of myxomatosis is a paper published in the British Journal of Experimental
Pathology in 1953, titled “The Pathogenesis of Infectious Myxomatosis; the Mechanism of Infection and the Im-
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munological Response in the European Rabbit (Oryctolagus cuniculus).” The study reported that the researchers
were able to make rabbits display the symptoms of myxomatosis (including death) by injecting them with “virulent
myxomatosis.” The recipe for this witches’ brew included ground-up organs and heated blood of sick, flea-bitten rab-
bits, “passed through” other rabbits, which were then bled to death to obtain the serum and then grown on chicken
embryos. Healthy rabbits injected—often several times—with this “virulent myxomatosis” usually do sicken and die.
The Wikipedia entry for “myxomatosis” includes a discussion of how to diagnose this so-called viral disease and
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hints at the problems involved. According to the entry, a myxomatosis diagnosis usually follows a description of the
“characteristic clinical appearance”—in other words, you can tell whether a rabbit has it by the symptoms. For further
confirmation, however, researchers have turned to three other techniques: histopathology, electron microscopy and
“virus isolation.” As regards the first, Wikipedia states, “Histopathologic examination of affected skin typically shows
undifferentiated mesenchymal cells within a matrix of mucin, inflammatory cells, and edema. Intracytoplasmic inclu-
sions may be seen in the epidermis and in conjunctival epithelium.” In other words, researchers do not actually see

20 Wise Traditions WINTER 2020